Some Observations on the Metabolism
نویسنده
چکیده
For many years clinical interest in D-galactose centered about the usefulness of galactose tolerance tests in the diagnosis of impaired liver function. Several investigators, employing an intravenous galactose tolerance test, have reported their results (1-3). The monograph by Stenstam (4) is perhaps the most extensive analysis of both the oral and intravenous tolerance test yet reported. In all of these studies the parameter for measurement of galactose metabolism has been the disappearance rate of the sugar from blood or the urinary excretion of the sugar for an arbitrary time interval. More recently research in galactose metabolism has had as its focal point the disease, congenital galactosemia. Kalckar and Maxwell, and their associates (5-7) have contributed to the delineation of the pathways of galactose metabolism and have pinpointed the enzymatic defect in galactosemia (8, 9). Other investigators have reported that certain steroids as well as menthol are able to prevent the depression of galactose oxidation in liver slices and homogenates caused by the metabolism of ethanol and aldehydes (10-12). The administration of progesterone (13) and menthol (12) has enhanced the ability of galactosemic subjects to oxidize galactose to CO. Because of the latter experiments it became important to delineate the physiologic disposition and metabolic fate of C14-galactose in normal subjects. This report presents the results of experiments performed for this purpose. Observations have been made of the effect of progesterone. menthol. and ethanol on galactose-1-C'4 oxidation.
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